100 Bilder zum Thema "qrs wave" bei ClipDealer

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Male, 60 years old, clinically diagnosed as acute extensive anterior wall myocardial infarction. The patient died of ventricular fibrillation after admission.
Ventricular tachyarrhythmia includes many clinical types, some benign and some malignant. For malignant ventricular arrhythmias, patients are at risk of death.
Bidirectional ventricular tachycardia is a kind of malignant arrhythmia. The polarity of QRS main wave alternates from beat to beat, and it is easy to degenerate into ventricular fibrillation.
During left posterior fascicular block, the ECG showed right axis deviation. The QRS wave in leads I and aVL was rS wave, and the duration of QRS wave was less than 120 ms.
The illustration shows the two patterns of ventricular tachycardia episodes.The green circle represents sinus rhythm. Picture A shows paroxysmal episodes of ventricular tachycardia, and picture B shows short bursts.
Male, 60 years old, clinically diagnosed as acute extensive anterior wall myocardial infarction. The patient died of ventricular fibrillation after admission.
Torsade de pointes refers to the pleomorphic ventricular tachycardia that occurs in the background of long QT interval, and the polarity of QRS wave twists around the equipotential line.
Note that the V3 lead of this ECG shows that the amplitude of R wave is greater than the amplitude of S wave, and there is counterclockwise rotation.
The QT interval of ECG is from the beginning of QRS wave to the end of T wave, representing the total time of ventricular depolarization and repolarization.
Sometimes, because the QRS axis is in the upper left quadrant, the high-amplitude R wave of left ventricular hypertrophy occurs in the limb leads, and left chest leads is normal.
A 36 year old man survived CPR after sudden syncope. The electrocardiogram was suggestive of Brugada syndrome type 1. Implantation of ICD therapy.
During the onset of variant angina pectoris, ECG is divided into non fusion wave, partial fusion wave and complete fusion wave according to the fusion degree of QRS wave, ST segment and T wave.
Electromechanical separation is a kind of terminal ECG. The patient's ECG has electrical signals, the ECG wave is widened with morphological abnormalities, and the ventricle has no contraction.
A 14-year-old leukemic child had a sudden wide QRS tachycardia with a frequency of 167 bpm, and the rhythm was regular. After anti-arrhythmia treatment, the patient recovered to sinus rhythm.
In case of acute anterior myocardial infarction, the characteristics of ST segment elevation in ECG can be used to deduce whether the culprit vessel system is the left main trunk or the proximal LAD.
R wave greater than S wave is judged to be positive; R smaller than S is judged to be negative; R equal to S amplitude is judged to be equipotential.
Female, 51 years old, diagnosed with mitral stenosis. When this ECG was taken, the patient still maintained sinus rhythm.Note that the P wave duration was widened.
A patient with acute extensive anterior myocardial infarction developed ventricular tachycardia during hospitalization and quickly experienced cardiac arrest.
At present, there is a younger trend in patients with acute myocardial infarction, so it is important to check the ECG for acute chest pain in young people.
In complete left bundle branch block, the conduction of the LBB can be completely interrupted or can still be conducted, but it is delayed by at least 45ms than the RBB.
Coronary artery spasm causes transmural myocardial ischemia, and ST segment elevation in ECG has localization characteristics. Criminal vessels can be derived from ST segment elevation leads in ECG.
Due to the large mass of the left ventricle, the dominant excitation potentials of the left and right ventricles are oriented towards the left ventricle, i.e. towards the left, Inferior and posterior.
It is best to measure the QRS wave duration in a 12 lead synchronous electrocardiogram, as some of the QRS wave start and end points are located on the isoelectric line.
Relative bradycardia refers to a pathophysiological phenomenon in which the patient's body temperature rises, but the pulse does not increase, which is common in some infectious diseases and jaundice.
A normal electrocardiogram includes normal morphology, amplitude, and various measurements of duration and interval, normal electrical axis, and normal R wave progression.
Electric impulses can be conducted, but the conduction speed slows down, resulting in conduction delay and affecting the morphology of the P wave, PR interval, and QRS wave.
Male, 84 years old, admitted to hospital with chest pain for 1 day. ECG showed acute inferior and posterior MI and possibly right MI. The patient died of ventricular fibrillation the next day.
The transverse vectorcardiogram generates a chest leads electrocardiogram, with the maximum ventricular excitation potential oriented towards the left posterior region.
When the rhythm of the atria originates in the lower part of the atria, the whole atria are excited from inferior to superior, producing negative P waves in the inferior leads.
Male, 84 years old, admitted to hospital with chest pain for 1 day. These ECG rhythms are the Holter monitor records of the patients after admission, and they are third degree atrioventricular block.
The 4-phase membrane potential of sinoatrial node pacing exhibits spontaneous depolarization, while the 4-phase membrane potential of ventricular myocytes remains stable.
On the conventional 12-lead ECG, under normal circumstances, there are some inherent patterns of QRS waves in different leads, which are not exactly the same.
In the spatial anatomy of the heart, the axis from the base of the heart to the apex of the heart is called the long axis, that is, the upper right side faces the lower left side.
The terminal excitation of the ventricle forms the final part of the S wave in lead V1, gradually returning to the isoelectric line, and forms a small S wave in lead V5.
In humans, Purkinje fibers are not distributed throughout the entire ventricular wall, but rather in the superficial myocardium beneath the endocardium and do not reach the epicardium.
When emphysema occurs, the diaphragm moves downwards, pulling the right atrium, causing an increase in the longitudinal longitude of the right atrium, and an increase in the amplitude of the sinus P wave.
QRS wave is a ECG wave generated by ventricular excitation, typically in a three-phase waveform, named qRs wave. The QRS waveform of each lead is different.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract forms a high amplitude R wave in the inferior leads and a QS wave or rS wave in the V1 lead.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract is a benign ventricular tachycardia. This ECG shows a short burst pattern of ventricular tachycardia.
When the first degree interatrial block occurs, the conduction time from the right atrium to the left atrium is prolonged, the P wave widens, and bimodal P wave ECG changes appear.
Male, 71 years old, was clinically diagnosed with upper gastrointestinal bleeding. During sleep at night, ECG monitoring showed sinus bradycardia, blood pressure 115 and 70mmHg.
When the frontal QRS axis is at +83, the R amplitude of lead aVF is the highest.The frontal QRS axis is almost perpendicular to the axis of lead .
Clockwise rotation electrocardiogram refers to the transition of the rS waveform of the chest lead to the left chest lead, with the transition lead exceeding the V4 lead.
Abnormal ECG refers to changes in depolarization waves and or repolarization waves, most of which are pathologic and few are physiological.
QRS wave is a ECG wave generated by ventricular excitation, typically in a three-phase waveform, named qRs wave. The QRS waveform of each lead is different.
When sinus bradycardia is obvious, the ventricle can be controlled by junctional escape and ventricular escape, and escape rhythm appears.
The initial excitation of the ventricle forms a small r wave in lead V1 and a small q wave in lead V6.
In ST segment elevation myocardial infarction, the ST-T of ECG will undergo a characteristic evolution process, and finally appear pathological Q wave, sometimes lasting for a lifetime.
In acute myocardial ischemia, the amplitude of T wave is increased first, and then the ST segment is elevated. When the end of QRS wave is deformed, there is a lack of collateral circulation.
Surrounding the atrioventricular ring, except for the anterior septum of the left ventricle, there is no distribution of accessory pathways, and accessory pathways can exist in other parts.

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