175 Bilder zum Thema "electrocardiogram education" bei ClipDealer

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When there is a the first degree interatrial block, the impulse from the right atrium is slowly transmitted to the left atrium, causing widening notched P wave.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract is a benign ventricular tachycardia. This ECG shows a short burst pattern of ventricular tachycardia.
Bidirectional ventricular tachycardia is a kind of malignant arrhythmia. The polarity of QRS main wave alternates from beat to beat, and it is easy to degenerate into ventricular fibrillation.
The illustration shows the two patterns of ventricular tachycardia episodes.The green circle represents sinus rhythm. Picture A shows paroxysmal episodes of ventricular tachycardia, and picture B shows short bursts.
The left main coronary artery can be divided into the left anterior descending artery and the left circumflex artery, and sometimes the intermediate branch artery.
Due to the large mass of the left ventricle, the dominant excitation potentials of the left and right ventricles are oriented towards the left ventricle, i.e. towards the left, Inferior and posterior.
A normal electrocardiogram includes normal morphology, amplitude, and various measurements of duration and interval, normal electrical axis, and normal R wave progression.
Electric impulses can be conducted, but the conduction speed slows down, resulting in conduction delay and affecting the morphology of the P wave, PR interval, and QRS wave.
Male, 23 years old, healthy. When the initial excitation potential of the ventricle deviates from a certain lead axis, a Q wave will be projected onto that lead, which is a physiological Q wave.
When acute left main artery occlusion causes ST segment elevation myocardial infarction, it is often accompanied by extensive anterior and high lateral myocardial infarction.
The transverse vectorcardiogram generates a chest leads electrocardiogram, with the maximum ventricular excitation potential oriented towards the left posterior region.
When intermittent conduction dysfunction occurs in the Bachmann bundle, intermittent left atrial abnormality may be seen on the ECG, which can be differentiated from anatomical left atrial enlargement.
When the rhythm of the atria originates in the lower part of the atria, the whole atria are excited from inferior to superior, producing negative P waves in the inferior leads.
Under normal circumstances, in the chest lead electrocardiogram, the amplitude of the R wave gradually increases from lead V1 to lead V6.
The terminal excitation of the ventricle forms the final part of the S wave in lead V1, gradually returning to the isoelectric line, and forms a small S wave in lead V5.
Idiopathic ventricular tachycardia originating from the right ventricular outflow tract is a benign ventricular tachycardia. This ECG shows a short burst pattern of ventricular tachycardia.
When the first degree interatrial block occurs, the conduction time from the right atrium to the left atrium is prolonged, the P wave widens, and bimodal P wave ECG changes appear.
In the aVR lead, the QRS wave can be in the form of QS, rS, Qr, rsr, etc., with the main wave being negative.
Second degree rinteratrial block includes two types: type 1 block with progressive worsening of conduction and interruption of conduction, and type 2 block with fixed conduction and interruption.
Some patients with severe sinus bradycardia have triggers that can disappear after treatment, while others are permanent and require treatment with ventricular pacemakers.
Septal q wave loss refers to the initial q wave loss of leads I, aVL, V5, and V6, which can be partially or completely lost.
The initial excitation of the ventricle forms a small r wave in lead V1 and a small q wave in lead V6.
When Bachmann bundle conduction is interrupted, the right atrium excites the left atrium through the coronary sinus, producing positive and negative biphasic P-waves in the inferior leads.
When Bachmann bundle conduction is interrupted, the right atrium excites the left atrium through the coronary sinus, producing positive and negative biphasic P-waves in the inferior leads.
Female, 6 years old, clinically diagnosed with Tetralogy of Fallot. The electrocardiogram shows sinus rhythm, extreme right axis deviation, biatria abnormality, right ventricular hypertrophy, etc.
Third degree block refers to persistent or permanent interruption of conduction, which can occur in any part of the conduction system and produce corresponding electrocardiogram changes.
The high lateral lead group includes leads I and aVL, used to explore the myocardium of the high lateralwall of the left ventricle.
During normal ventricular excitation, the earliest epicardial breakthrough point is located in the paraventricular septal area, and the RV outflow tract and the base of the LV are finally excited.
A 2:1 left bundle branch block is considered when complete left bundle branch block alternates with normal QRS complexes and the PR interval is fixed.
Under physiological conditions, the upward sloping ST segment depression forms a U-shaped curve, where the depressed PR segment, J point, and ST segment form a smooth parabola.
Under normal circumstances, when the initial excitation potential of the ventricle is far away from a certain lead axis, a Q wave will be projected on that lead, which is a physiological Q wave.
Electric impulses can be conducted, but the conduction speed slows down, resulting in conduction delay and affecting the morphology of the P wave, PR interval, and QRS wave.
During ventricular depolarization, a spatial QRS loop is generated, projected onto the frontal lead system, forming the frontal QRS loop, which further forms the electrocardiogram.
Under normal circumstances, notch T waves are more common in leads V2-V3 and are caused by asynchronous local ventricular repolarization.
In the aVR lead, the QRS wave can have an initial r wave or no initial r wave, depending on whether the initial excitation is directed upwards or downwards.
In a wide QRS rhythm, the nature of the P wave can be observed in leads with lower QRS wave amplitudes. In this case, retrograde P waves can be seen in leads aVR and aVL.
In frontal lead system, when the average ventricular excitation potential is in the range of +60 degrees to+90 degrees, the QRS main wave of aVL lead is negative.
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In a wide QRS rhythm, the nature of the P wave can be observed in leads with lower QRS wave amplitudes. In this case, retrograde P waves can be seen in leads aVR and aVL.
In the frontal plane lead system, when the maximum QRS wave potential is more parallel to a certain lead, the R wave amplitude of that lead is highest in the limb lead.
When premature contractions occur, the ventricular filling time is shortened and the ventricular filling volume decreases, which can lead to a decrease in QRS wave amplitude.
If the electrocardiogram during the onset of ventricular tachycardia cannot be recorded, it is possible to incorrectly analyze the QRS waveform based on the QRS waveform during the attack.
When acute left main artery occlusion causes ST segment elevation myocardial infarction, it is often accompanied by extensive anterior and high lateral myocardial infarction.
heart rhythm ekg note on paper Doctors use it to analyze heart disease treatments
eart beats cardiogram
Sometimes, left ventricular hypertrophy with tall T waves is easily misdiagnosed as hyperkalemia and hyperacute T waves, and ECG needs to be carefully identified in combination with clinic.
Surgeons team working with monitoring of patient in surgical operating room,Doctor operation in operation room at hospital concept.
Torsade de pointes refers to the pleomorphic ventricular tachycardia that occurs in the background of long QT interval, and the polarity of QRS wave twists around the equipotential line.
Four anatomical malformations of tetralogy of Fallot: 1 aortic straddling; 2 ventricular septal defect; 3 right ventricular hypertrophy and 4 pulmonary artery stenosis.
Congenital heart disease: Tetralogy of Fallot

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